Researchers at Stanford University School of Medicine have found that an inflammatory metabolite in the body, typically suppressed with pain relieving and anti-inflammatory drugs like aspirin or ibuprofen, expedites the healing process for muscles by activating stem cells. The metabolite, called prostaglandin E2 (PGE2), is part of the body’s normal inflammatory response, but painkillers usually taken to relieve muscle soreness inhibit its production. The discovery of this mechanism has given insight to scientists of the micro environment stem cells need to function in, paving the way for enhanced treatments for muscle injuries and trauma as well potential treatments to combat the natural degradation of muscle mass that accompanies aging.
Professor of microbiology and immunology, and director of Stanford’s Baxter Lab for Stem Cell Biology, Helen Blau, PhD stated that "When we tested the effect of a one-day exposure to PGE2 on muscle stem cells growing in culture, we saw a profound effect on the proliferation of the cells. One week after a single one-day exposure, the number of cells had increased six-fold compared with controls." This could mean that honing in on a treatment for muscles can soon incorporate methods that do not inhibit but rather stimulate the work of PGE2 in muscle regeneration.
To learn more about stem cells, and how families can bank their own valuable stem cells by recovering the very powerful dental pulp stem cells during routine dental procedures; such as wisdom teeth extractions or the during the loss of baby teeth, visit StemSave or call 877-783-6728 (877-StemSave) today.
The Future of Regenerative Medicine is Now™
To view the full article, click here.